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Magnetic therapy for gout helps relieve its painful symptoms and make attacks less frequent and intense. With gout, metabolism is disrupted, uric acid is poorly excreted from the body, and crystals of its salts are deposited in the joints. The result is inflammation, severe pain, and in severe cases, destruction of the joint and impaired mobility.
Magnetic therapy for gout is indicated in combination with diet and medications.
Magnetic therapy for gout gives the following results:
- relieves both sharp and aching pain;
- improves blood supply to joints;
- accelerates the utilization of uric acid salts;
- improves metabolic processes in cells;
- reduces the inflammatory response;
- promotes restoration of damaged tissues.
Regular magnetic physiotherapy procedures will help stop the progression of gout, prevent irreversible changes and preserve joints.
Magnetic therapy for gout of the legs will reduce pain when walking and help restore mobility. Magnetic therapy devices are universal and can be useful for treating a variety of diseases. Home appliances are designed specifically for use by people without medical education; they are affordable and easy to use. Gout is a chronic disease, so in case of gout it is convenient to have your own magnetic therapy device.
Devices for magnetic therapy of gout
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Treatment of gout
The goal of gout treatment is to reduce pain and prevent recurrent attacks and complications (damage to joints and urolithiasis). The following treatment methods are used:
Diet. If you are overweight, you need to reduce your caloric intake. In addition, significantly reduce the presence of meat products and seafood in the diet. Reduce your consumption of alcohol and especially beer.
Drug treatment. There are drugs that increase the secretion of uric acid (uricosuric drugs - for example benemid), reducing the production of uric acid (uricodepressive drugs - for example allopurinol), drugs that relieve an acute attack - colchicine, NSAIDs, corticosteroids.
Three stages of gout
Stage 1: High levels of uric acid in the blood
- Blood uric acid levels may be elevated, but there are no clinical manifestations.
- High levels of uric acid in the blood (hyperuricemia) may not progress, and gout symptoms may not appear at all.
- In some people, the primary manifestation may be kidney stones rather than a gout attack.
Second stage: Episodes of acute gouty arthritis alternate with periods without symptoms.
- This stage is also called interictal.
- Uric acid crystals begin to form in the synovial fluid, usually in one joint (the big toe) - and the body responds with an inflammatory response and an attack episode occurs.
- Although the thumb is most commonly affected, it is possible that other joints may also be involved. Although the big toe is the most common site for a gout attack, gout can develop in other joints.
- After an attack of gout ends, the damaged joint quickly returns to normal within 2-3 days before the next attack (as a rule, the attack must be repeated within 1-2 years).
- Over time, the intervals between attacks become shorter and the intensity of symptoms becomes more pronounced. In addition, other joints are also involved in the process.
Third stage: Chronic tophi gout
- If the symptoms of gout appear occasionally and treatment is not carried out for several years, then a chronic process occurs and other joints are involved. The intervals between attacks disappear. This stage of gout often resembles other types of arthritis (usually osteoarthritis).
- By this time, sufficient accumulation of uric acid crystals has occurred to form nodules (tophi) in various parts of the body. Tophi are dense to the touch, mobile and located under the skin. The underlying skin may be red and thinned (the color of tophi may be yellow or cream).
- As a rule, tophi are localized near the elbow or near the toes, hands, and auricle. With progression, tophi may develop near the joints or in the cartilage tissue of the auricle. Tophi located in cartilage tissue can lead to cartilage destruction.
Gout
Gout is a painful and potentially disabling form of arthritis associated with a metabolic disorder in which the concentration of uric acid in the blood is elevated, primarily causing painful inflammation in the joints. About 80 percent of gout patients are men. The disease usually affects them between the ages of 40 and 60, less often at a younger age. Women usually don't develop gout until after menopause.
Specialists at the Miracle Doctor clinic diagnose and treat patients with both acute and chronic manifestations of gout. The clinic has modern equipment for conducting laboratory and instrumental research.
Symptoms
The first symptoms are usually severe attacks of painful swelling of individual joints, in half of the cases - the metatarsophalangeal joint of the big toe. The swelling may be red and hot. A direct trigger for pain is often heavy food or alcohol consumption.
A typical “textbook case” that suggests a high likelihood of an acute attack of gout is as follows: sudden, extremely painful inflammation of the metatarsophalangeal joint in a 40-year-old man who had been celebrating the night before with a lot of food and alcohol.
How it develops
An imbalance in the formation of uric acid in the body and its elimination leads to the development of the disease hyperuricemia - an excess amount of uric acid in the body. Actually, the formation of uric acid is associated with the breakdown of purines in the intestine - chemical compounds contained in the cells of the body. But they also enter the body with foods consumed.
This results in the formation of urate crystals in the form of monosodium urate or uric acid, which can accumulate in joints, joint capsules and tendons. They lead to painful inflammation and, if left untreated, joint damage. Crystals can also settle in the kidneys and cause kidney stones.
Reasons for development
- Most gout sufferers have an innate tendency to have high uric acid levels.
- Overweight.
- Eating too much and drinking too much alcohol
- Lack of exercise.
- By the way, according to information from foreign sources, fructose increases the risk of gout, as it suppresses the excretion of uric acid by the kidneys, just like alcohol. Even one sweet drink containing fructose per day, such as fruit juice, increases the risk of gout in healthy people by 45 percent. Therefore, anyone who already suffers from gout should avoid drinks and foods containing fructose.
Diagnostics
The symptoms and signs of an acute attack of gout are often very characteristic, so it is usually not difficult for a doctor to make the correct diagnosis.
Laboratory Tests Your current uric acid level can be determined using a blood test. However, this is not always noticeable during an acute attack, since uric acid levels can quickly return to normal levels. Repeated uric acid tests are more informative.
In unclear cases, joint puncture and examination of the synovial fluid, in which uric acid crystals can be seen under a microscope, help confirm the diagnosis.
X-ray examination
may show typical joint changes in advanced gout.
Forms of gout
Doctors distinguish between two forms of gout:
Primary gout is a congenital disease
Most people with gout suffer from an inborn error of metabolism. Doctors then talk about “primary hyperuricemia” or “primary gout,” when the kidneys do not remove the normal amount of uric acid from the body.
In rare cases, the body produces so much uric acid that the kidneys become overworked. The cause is a genetic defect, the so-called Lesch-Nyhan syndrome. Occurs predominantly in boys.
Acquired (secondary) gout
In so-called secondary gout, other diseases cause excess uric acid. With leukemia, for example, the body's own cells die en masse. This releases a large amount of purines, which accumulate in the blood.
Other diseases that cause increased uric acid production include:
- some types of cancer;
- anemia;
- some drugs (cytostatics);
- radiation therapy as part of cancer treatment;
- kidney diseases;
- untreated or poorly controlled diabetes.
Exacerbation of gout
An acute attack of gout occurs when uric acid levels exceed a certain level. Main triggers:
- excessive consumption of foods rich in purines, such as meat and organ meats.
- Excessive consumption of foods rich in fructose, such as sweetened fruit juices.
- too much alcohol: Alcohol also increases uric acid levels. This is especially true for beer, which is also particularly high in purines.
- Crash Diets: To gain energy, the body breaks down muscle through crash diets. In this case, many purines are released.
- physical overexertion: lactic acid is formed in the body, which is excreted by the kidneys and thereby blocks the breakdown of uric acid.
- diuretics or laxatives: they thicken the blood when used excessively or for a very long time. This increases the concentration of uric acid.
Gout treatment
After an acute attack of gout, the goal of treatment is to relieve pain and stop inflammation as quickly as possible - for example, with so-called non-steroidal anti-inflammatory drugs (NSAIDs) such as ibuprofen and diclofenac.
Once symptoms subside, long-term therapy can prevent progression of gout and further gout attacks. It is recommended when the patient experiences more than two attacks of gout per year, there are kidney stones accompanying gout, if gout has led to further changes in the body. However, gout cannot be cured with medications. Once the drugs are stopped, their effect on uric acid levels is lost and they rise again.
Losing excess weight will automatically reduce uric acid levels, but weight loss should be slow and controlled. Strict fasting can trigger an acute attack of gout!
Drink plenty of fluids - nutritionists recommend drinking at least two liters a day, preferably mineral water or unsweetened tea. Fluids in the body help keep uric acid concentrations low and support the filtering function of the kidneys. Water also flushes out uric acid and reduces uric acid levels.
Controlled physical activity - movement has a positive effect on joints with gout. Function improves and inflammatory symptoms resolve more quickly. However, if you have gout, you should not overwork yourself - the lactic acid that forms slows down the breakdown of uric acid through the kidneys.
Diet for gout
Changing your diet plays a decisive role:
- + Foods rich in purines can only be consumed in small portions: among other things, purines are present in the genome of all living cells. When it is broken down, uric acid is formed. This applies both to the patient’s own overripe cells and to food. Foods rich in purines include meat (especially organ meats), sausages, seafood and some types of fish.
- +As little alcohol as possible: Excessive alcohol consumption is especially problematic if you have gout. Its breakdown products are excreted through the kidneys and compete with uric acid. Alcohol slows down the breakdown of uric acid and increases its level. Even if the patient drinks alcohol only occasionally, this can trigger a gout attack. It is especially important to avoid beer consumption, since in addition to alcohol, it is also high in purine.
- +You should be careful with fructose: fructose is not only found in fruits. It is also used to sweeten juices, yogurts and other foods. The breakdown of fructose in the body increases the production of purines. At the same time, like alcohol, sugar suppresses the excretion of uric acid through the kidneys.
- +Fat also prevents the excretion of uric acid. Therefore, gout patients should eat as little high-fat foods as possible. If possible, avoid more than 30 percent of your daily calories from fat. This limit is reached quickly because fat has the highest energy capacity of all nutrients.